Low-calorie diet can cause changes in the activity of the body at the molecular level, drowning out the activity of proteins associated with aging. That is the conclusion drawn by researchers from the United States, summarizing study involving roundworms, mice and insects.
Scientists from the University of Utah studied kinase TOR have roundworms kind of C. Elegans. Scientifically it has been proven that a compound of TOR protein associated with aging and disease. TOR activity leads to age deterioration of the body and serves as a factor in the development of several chronic diseases, including diabetes, cardiovascular disease, cancer and neurodegenerative diseases like Alzheimer's in and Parkinson's disease. Too high a level of TOR leads to insulin resistance and fat accumulation. Moreover, TOR regulates cell proliferation plays an important role in cancer
"Our work has shown that the gene TOR depends on a second gene, the so-called pha4 / FoxA, which also controls the aging process", - commented on the results of his study Head Susan Mango.
Mango and his colleagues found that eating high-calorie foods unnecessarily stimulates the activity of the gene TOR, which inhibits the action of a gene pha4 / FoxA. As a result, the life expectancy is reduced.
On the contrary, the loss of activity due to the low-calorie diet TOR aging worm C. Elegans slowed. (READ MORE)
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